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Beyond obvious effects on energy balance, dietary quality can also influence obesity risk through changes in metabolic adaptation due to a high refined carbohydrate diet, in resting energy expenditure (such as through brown or beige adipose tissue thermogenesis), in microbiome calorie utilization (with corresponding greater or lesser utilization by the host tissues), and in epigenetic or trans-generational risk of obesity [87,88]

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Select BAs, CA, GCA and taurocholic acid (TCA) were evaluated in the serum from rats dosed with various hepatotoxicants, including APAP and nonhepatotoxicants (Luo et al