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HarteveldCLAchourAArkesteijnSJGTer HuurneJVerschurenMBhagwandien-BisoenSet alThe hemoglobinopathies, molecular disease mechanisms and diagnostics

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This gives rise to a vicious cycle: hepcidin levels in patients with -thalassemia are frequently too low, thereby triggering excessive iron absorption in the intestines and the mobilization of iron in macrophages, ultimately leading to iron overload in various organs, including the heart (5254)

RO can reduce cell apoptosis at the injury site by reducing ROS levels, but also alleviate the inflammatory response by increasing the ratio of M2/M1 microglia and inhibiting the expression of proinflammatory factors, thereby promoting nerve regeneration and functional recovery (Figure 9B) [185]