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These reactions can result in the formation of adducts, which can serve as biomarkers of oxidative stress and lipid modification

-MSH reduced leukocytes, mast cells, and fibroblasts at 3 and 7 days post-injury, while also decreasing scar area and improving collagen fiber organization at days 40 and 60, indicating its potential to promote a more regenerative wound healing pathway in the studied model

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Concurrently, ammonium metabolites exhibit mitochondrial toxicity, collapsing membrane potential, and depleting ATP, while nutrient stress activates AMPK to phosphorylate ULK1, initiating pro-survival autophagy (201, 202)