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Precisely at the core of the central gain model is the hypothesis that extensive spontaneous and sound-evoked neuronal hyperactivity in higher auditory structures (e.g., DCN) may constitute a key neural correlate underlying the sound percepts of tinnitus 46,47,48,49

But beneath the hype and fear is a simple story: these drugs target a core metabolic signaling pathway that influences cardiovascular health, inflammation, cognition, kidney function, and long-term resilience

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Because the manufacturers of these branded drugs were unable to meet demand, the FDA declared many dosage forms in shortage