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Markers of apoptosis, including elevated Bax and caspase-3 along with reduced Bcl-2, were significantly dysregulated in the STZ-induced diabetic group in this study, indicating enhanced pro-apoptotic signaling and mitochondrial dysfunction 55,56

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All work is non-clinical and mechanistic in nature

In our study, there were no significant differences in the expression levels of reduced or oxidized glutathione, regardless of administration of the Si-based agent