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Ethanol metabolism, CYP2E1 induction, compromised antioxidant defenses, mitochondrial injury, activation of Kupffer and stellate cells, hypoxia, and iron overload can all contribute to the alcohol-induced oxidative environment

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that model OCR output is insensitive to the inclusion of the comproportionation kinetics ( R 5 ) and finally

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Environ Monit Assess

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Footnotes 12 Abbreviations used: IUGR, intrauterine growth retardation

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