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Demonstrates positive regulatory effects on genes related to antioxidant enzymes, growth factors, and anti-inflammatory signaling [3] [4]

Taken together, these mechanistic and clinical threads support a model in PCOS whereby platelet activation and PMP release prime neutrophils for NETosis, and NETs provide a prothrombotic scaffold that recruits and activates additional platelets and coagulation factors, producing an escalating thrombo-inflammatory loop with potential consequences for ovarian microvasculature, endometrial receptivity, and long-term cardiovascular risk [77]

Nat Rev Urol (2026)

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