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While the peptide did not significantly alter mitochondrial or lysosomal activity, or the expression of senescence markers p16 and laminB1, its profound effects on reducing DNA damage and enhancing dendritic arborization led the authors to conclude that the EDR peptide partially protects human neurons from age-related deterioration and stimulates structural neuroplasticity

Critical Reviews in Analytical Chemistry, 46(5), 389399

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