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Colchicine also has a major role when patients begin therapy with allopurinol (see below) to prevent the increase in gout attacks that can happen when allopurinol is begun

While further research is needed to clarify the causal pathways, the potential role of autoimmunity as a driver of persistent symptoms should not be overlooked in clinical practice

You should never have to wonder if what you are putting into your body is doing more harm than good

Mitochondrial dysfunction in huntingtons disease: Pathogenesis and therapeutic opportunities

Reduction in the activity of this enzyme decreases the conversion of substrates into fatty acids, thereby shifting cellular metabolism toward lipid mobilization rather than storage [1]