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showed that ob/ob mice fed both high- trans -fat, high-fructose and high-cholesterol (AMLN) diet as well as a standard chow diet had the same level of CAT activity, while AMLN diet-fed mice had markedly increased oxidative DNA damage (Boland et al

a neutral amino acid transporter ASCT1, a cystine transporter xCT, enzymes for the transsulfuration pathway, and autophagy markers, were all elevated in the liver as the result of the AAP treatment, the AsA deficiency suppressed their induction

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