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In this study, accumulation of lipid peroxidation end products MDA and 4-HNE and intracellular iron coincided or preceded with the hepatocellular injury in the Aldh2*2 KI mice after AA exposure, suggesting that ferroptosis is involved in the exacerbation of liver injury [56,77]

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These observations indicate that detoxification capacity is not static, but rather represents a dynamic, inducible adaptive response