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By examining current evidence from cellular, animal, and clinical studies, this review aims to provide a detailed evaluation of sulforaphanes potential as a neuroprotective agent in neurological disease treatment and prevention

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Signaling pathways from Trk activation: PI3K/Akt pathway phosphorylates Bad (pro-apoptotic protein) preventing it from blocking Bcl-2 and Bcl-xL (anti-apoptotic proteins)