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Intestinal barrier disruption and dysregulated mucosal immunity contribute to kidney fibrosis in chronic kidney disease

While it shows potential for promoting tissue repair and reducing inflammation, its premature to label it a cure for chronic conditions without more robust clinical data

Nitro-oxidative stress and inflammatory response, with the consequent activation of TRP channels, seem to play a major role in the beginning and development of NP

is supported by the Canada Research Chair Program, a BBDC-Novo Nordisk Chair and CIHR grant 123391 and 154321

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