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63 Prolonged skeletal muscle inactivity promotes mitochondrial dysfunction, resulting in increase in mitochondrial ROS production and leading to protracted oxidative stress and disturbed redox signaling, 64 which can exacerbate IR through multiple pathways such as JNK and NF-B pathways (Fig

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TIME COURSE EVALUATION OF LACOSAMIDE ALONE AND IN POLYPHARMACY ON BEHAVIORAL MANIFESTATIONS AND OXIDATIVE STRESS IN LITHIUM-PILOCARPINE-INDUCED MODEL INTRODUCTION Temporal lobe epilepsy (TLE) is a chronic neurological disorder characterized by the frequent incidences of spontaneous recurrent seizures (SRSs) and 30% of individuals show pharmacoresistance with the typical available anti-seizure drugs (ASDs) (1)