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Moreover, peroxidative ROS levels depend on the caution of the antioxidant systems, which shield cells from oxidative harm, incorporate various peroxisomal enzymes, such as catalase (CAT), superoxide dismutase (SOD), glutathione peroxidase (GPX), and peroxiredoxins (PRDX), as well as low molecular weight non-enzymatic antioxidants [17]

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Studies suggest this rebalancing occurs through effects on thymic peptide signaling and restoration of neuroendocrine-immune system communication that deteriorates with aging