It happens because Semaglutide mimics a hormone called GLP-1 (glucagon-like peptide-1), which helps regulate appetite and slows digestion in the first week or two
Thats why our White Plains team focuses on gradual transitions, metabolic support, and strategies designed to help patients maintain their results
Hormones & Signaling GLP-1: released after eating Insulin: blood sugar regulation Glucagon: raises blood sugar when it drops Oxytocin: bonding, labor, social behavior Vasopressin: blood pressure and water retention Pain & Mood Endorphins: your natural painkillers, released during exercise Enkephalins: pain modulation throughout the nervous system Dynorphins: stress response and pain regulation Immune Defense Defensins: antimicrobial peptides that attack bacteria, fungi, and viruses Cathelicidins: front-line immune response in skin and lungs Digestion Cholecystokinin (CCK): signals fullness after eating Ghrelin: the hunger hormone, signals when you need to eat Secretin: regulates stomach acid and pancreatic function Cardiovascular BNP (B-type natriuretic peptide): regulates blood pressure
Delayed Gastric Emptying A condition called gastroparesis, where the stomach empties food more slowly than normal, can contribute to GERD
Appetite Regulation: The delay in gastric emptying also has an impact on appetite signals, which are partly regulated by how quickly the stomach empties its contents
