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This further activates the intracellular NF-B signaling pathway and the Janus kinase 2/signal transducers and activators of transcription 3 (JAK2/STAT3) pathway, thereby inhibiting the production of pro-inflammatory cytokines such as IL-1 and TNF-, ultimately exerting anti-inflammatory effects (99)

& Wolff, J

Consequently, mutations in TP53 lead to a loss of this repression, resulting in sustained SLC7A11 activity and greater cellular resistance to ferroptosis (Leu et al., 2019)

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How compartment-specific GSH metabolism modulates GPX4 activity during ferroptotic stress remains an important unresolved question